Buy Melanotan-2 (MT-2) – 99%+ Pure, COA Verified

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Certificate of Analysis Janoshik Analytical report #150246 Tested Apr 2026. Published online, not packed with orders.

Measured purity 99.866% Result Janoshik Analytical reported for the sample tested.

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Form Lyophilised powder Supplied in sealed vials. Storage conditions are listed in the specifications.

Testing history 2 reports on file Earlier certificates stay published with their own test dates.

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Melanotan-2

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What It's Researched For

In plain terms, Melanotan-2 is studied for skin tanning plus libido and appetite, because it hits several melanocortin receptors at once. Here is what that looks like across the research.

Tanning & skin pigment

Research explores how it darkens the skin by switching on natural pigment production, with little or no sun needed. Small early human studies and animal work both look at this.

Libido & sexual function

Animal studies and early human research investigate its effect on arousal and sexual function, the brain pathway that later inspired the FDA-approved PT-141.

Appetite & body weight

Animal studies look at reduced food intake and changes in body composition through the brain's appetite circuits.

Melanocortin biology tool

Used in research as a benchmark compound for mapping how the melanocortin receptor family works.

Scientific Overview

In short: Melanotan-2 (MT-2) is a cyclic non-selective melanocortin receptor agonist studied for UV-independent skin pigmentation, appetite modulation, and sexual arousal research.

Key research facts

MC1R agonism activates cAMP-PKA-CREB-MITF cascade for tyrosinase-driven eumelanin biosynthesis

MC4R activation in hypothalamic nuclei modulates sexual arousal through central neuroendocrine pathways

Appetite suppression via MC4R-mediated anorexigenic signaling in arcuate nucleus neurons

Crosses blood-brain barrier due to cyclic lactam structure and lipophilic modifications

Preserves His-D-Phe-Arg-Trp melanocortin pharmacophore with enhanced proteolytic stability over linear α-MSH

These points summarise lab themes, not human outcomes.

Melanotan-2 is a lab-engineered shortened and cyclised version of α-MSH, the natural hormone your body uses to trigger tanning. The modifications make it far more resistant to enzyme breakdown and much more potent than the natural molecule at binding all four melanocortin receptors.

Because MT-2 is non-selective, it hits the receptors that control pigmentation (MC1R), sexual arousal and appetite (MC4R), and exocrine function (MC5R) at the same time. In research this produces the characteristic triple signature: darker pigmentation, reduced food intake, and upregulated libido in animal models.

Unlike its selective cousin afamelanotide (Melanotan-1), MT-2 is not approved for any human therapeutic use. It is a research compound only, and its non-selective profile is part of why bremelanotide (PT-141) was later developed as a more targeted MC4R agonist.

Melanotan II is a synthetic lactam-bridged cyclic heptapeptide (Ac-Nle-c[Asp-His-D-Phe-Arg-Trp-Lys]-NH₂, MW 1024.18 Da) derived from truncation and cyclization of α-MSH, preserving the His-D-Phe-Arg-Trp pharmacophore essential for melanocortin receptor activation. It functions as a non-selective agonist of MC1R (melanogenesis), MC3R (energy homeostasis), MC4R (sexual function and appetite), and MC5R (exocrine secretion), crossing the blood-brain barrier to exert central neuroendocrine effects (Hruby et al., Journal of Medicinal Chemistry, 1995). MC1R activation stimulates eumelanin biosynthesis through cAMP-PKA-CREB-MITF transcriptional cascading, while MC4R agonism in hypothalamic nuclei mediates both appetite suppression and pro-erectile/arousal signaling. Pharmacokinetic analysis in rats demonstrates IV t½ of 0.5-1.5 hours, Vss of 0.2-0.5 L/kg, and oral bioavailability of 4.6% (Ugwu et al., Pharmaceutical Research, 1994).

At a glance

Mechanism and receptors MT-2 binds all four melanocortin receptors, triggering melanin production in the skin and activating appetite and arousal circuits in the brain.

Experimental models Hruby et al. 1995, MT-2 design, rodent receptor binding and in-vivo pigmentation (J Med Chem); Ugwu et al. 1994, rat pharmacokinetics and oral bioavailability (Pharm Res).

Clinical research Dorr et al. 2004, small human trial of MT-2 + UV combined tanning response (Arch Dermatol); Wessells et al. 2000, human pilot assessing erectile response to MT-2 (Int J Impot Res).

Molecular characteristics Molecular weight 1024.18 Da; sequence Ac-Nle-c[Asp-His-D-Phe-Arg-Trp-Lys]-NH₂.

Evidence maturity Mixed (human + animal) — Mechanism is well-characterised in human and animal receptor pharmacology (Tier 1-2). Pigmentation and arousal endpoints have small human pilot studies (Tier 1). Long-term safety in research subjects is predominantly anecdotal (Tier 3). MT-2 itself is not FDA-approved; bremelanotide (PT-141, closely related) is approved for HSDD.

Analytical verification Batch analysis reports identity and purity for the sample tested. It does not establish sterility, endotoxin or heavy-metal status, pharmaceutical equivalence, or any clinical property.

Research limitations Published clinical findings describe the study materials and protocols used by investigators; they do not establish the effects or safety of this research material.

Research Video

Mechanism of Action

MT-2 binds all four melanocortin receptors, triggering melanin production in the skin and activating appetite and arousal circuits in the brain.

Pathway Effect Why it matters MC1R (skin) Activates cAMP/PKA/CREB/MITF to turn on tyrosinase Produces eumelanin, the dark pigment that provides UV protection MC4R (hypothalamus) Reduces food intake and triggers arousal signalling Drives both appetite suppression and pro-erectile effects MC3R (energy balance) Modulates nutrient partitioning and metabolism Contributes to body-composition changes seen in animal models MC5R (exocrine) Stimulates sebaceous and lacrimal secretion Explains facial flushing and oily skin sometimes seen in research protocols Cyclic lactam bridge Locks the peptide into an active conformation Protects against proteolytic breakdown and extends duration of action

Deeper dive for scientific readers

Hruby and colleagues (Journal of Medicinal Chemistry, 1995) designed MT-2 as a lactam-bridged cyclic heptapeptide preserving the conserved His-D-Phe-Arg-Trp pharmacophore. The D-Phe7 substitution confers protease resistance and superpotent receptor binding. Rat PK (Ugwu et al., Pharmaceutical Research, 1994) gives an IV t½ of 0.5-1.5 h, Vss 0.2-0.5 L/kg, and ~4.6% oral bioavailability. Because MT-2 crosses the blood-brain barrier, receptor activation is both peripheral and central - which is why nausea and facial flushing are the most commonly reported side effects in research.

Common Questions People Are Asking

What is Melanotan-2?

Melanotan-2 (MT-2) is a synthetic cyclic heptapeptide analogue of α-MSH (Ac-Nle-c[Asp-His-D-Phe-Arg-Trp-Lys]-NH₂, MW 1024.18 Da) that non-selectively activates the MC1R, MC3R, MC4R and MC5R melanocortin receptors. It is supplied as a lyophilised research-grade powder for laboratory use only - it is not approved for human use.

What does Melanotan-2 do?

In research models MT-2 activates MC1R to stimulate eumelanin synthesis (the pathway behind UV-independent pigmentation), and activates MC4R in the hypothalamus, which is associated with appetite suppression and arousal signalling. These are findings in animal/research models; New-U makes no human-use or cosmetic claims.

Does Melanotan-2 work without sun?

Because MT-2 stimulates melanocyte MC1R directly, melanogenesis can be triggered in research models with little or no UV exposure - unlike a normal tan that requires sunlight. Published observations note that UV exposure still substantially accelerates and deepens the studied pigmentation response. This is research context only, not a cosmetic or human-use recommendation.

Does Melanotan-2 change eye color?

There is no credible scientific evidence that MT-2 changes iris (eye) colour. The melanocytes it acts on are in the skin and hair; the documented pigmentation effects are darker skin and darkening of existing moles/freckles, not eye colour. Reported for research context only.

How is Melanotan-2 reconstituted?

Lyophilised MT-2 is dissolved in bacteriostatic water for multi-use research vials. Concentration in mg/mL equals the vial mass in mg divided by the millilitres of diluent added - e.g. a 10 mg vial reconstituted with 1 mL gives 10 mg/mL. Keep the reconstituted vial refrigerated at 1–6 °C and protected from light. This is laboratory preparation math only; no human-use dosing guidance is provided.

What is the difference between Melanotan-1 and Melanotan-2?

Melanotan-1 (afamelanotide) is a linear 13-amino-acid α-MSH analog that is more selective for MC1R and is EMA/FDA-approved as Scenesse for erythropoietic protoporphyria. Melanotan-2 is a smaller cyclic heptapeptide that activates MC1R, MC3R, MC4R, and MC5R non-selectively, producing broader effects and more side effects.

Why does Melanotan-2 cause nausea and facial flushing?

Those effects come from non-selective activation of MC4R and MC5R in the brain and exocrine glands. MC4R activation in hypothalamic nuclei drives both appetite suppression and nausea, while MC5R stimulates sebaceous secretion and vasomotor responses that show up as facial flushing.

How does Melanotan-2 relate to PT-141?

PT-141 (bremelanotide) is essentially an active metabolite of Melanotan-2 with a free carboxylic acid instead of an amide at the C-terminus. It is more selective for MC4R-mediated sexual arousal pathways and is FDA-approved as Vyleesi for generalised HSDD in premenopausal women.

How should Melanotan-2 be stored?

Keep lyophilised powder at −20 °C in the freezer. Once reconstituted with bacteriostatic water, refrigerate at 1-6 °C and protect from light. Reconstituted solutions are generally stable for several weeks under those conditions.

What Melanotan-2 dosage is described in research?

The research literature typically describes a low-microgram subcutaneous "loading" phase followed by smaller intermittent "maintenance" amounts, with melanogenesis driving the studied pigmentation response. New-U publishes this only as descriptive research context - it is not a dosing instruction.

What does Melanotan-2 before and after show in studies?

In pigmentation research, MC1R activation increases eumelanin synthesis, so studied endpoints describe a gradual darkening over roughly 1-4 weeks of repeated exposure that fades once dosing stops. Magnitude depends heavily on baseline skin type, amount and UV exposure. Reported for research context only - not a cosmetic or human-use claim.

How long does Melanotan-2 take to work and how long does it last?

Pigmentation is cumulative rather than immediate: pigmentation research describes a gradual darkening building over roughly 1-4 weeks of repeated low-dose exposure, then fading over weeks once dosing stops as melanocytes turn over. The peptide itself clears quickly (rodent IV half-life ~0.5-1.5 h) but the studied pigment effect outlasts the circulating compound. These are descriptive research-context observations, not human-use or cosmetic guidance.

What are the side effects of Melanotan-2 reported in research?

The literature most commonly reports nausea, facial flushing, appetite suppression and darkening of existing moles/freckles, arising from non-selective MC3R/MC4R/MC5R activation. New-U supplies Melanotan-2 as a research compound only and makes no safety or human-use claims; these are summarised research findings, not medical advice.

Is Melanotan-2 legal, and is it FDA-approved?

Melanotan-2 is not approved by the FDA, EMA or any major regulator for human use - only the related, more selective afamelanotide (Melanotan-1 / Scenesse) and bremelanotide (PT-141 / Vyleesi) hold approvals for specific indications. Melanotan-2 is sold in the US strictly as a research compound; buyers are responsible for compliance in their jurisdiction.

Where can I buy Melanotan-2?

Right here - Melanotan-2 is supplied directly by New-U Research Compounds on this page as lyophilised research-grade material, with an independent Janoshik Analytical Certificate of Analysis published on this page, and shipped direct from source worldwide in discreet, tracked packaging.

How much does Melanotan-2 cost?

Melanotan-2 pricing is shown live on this page, per pack size - sealed 10-vial research packs, or single vials where listed (an order needs at least 5 single vials in total, or any 10-vial pack). The batch Certificate of Analysis is published on this page, and shipping is free on orders over $350.

Is Melanotan-2 third-party tested?

Yes. Melanotan-2 has an independent Janoshik Analytical Certificate of Analysis, which reports 99.866% purity for the sample analysed. The report number and test date are on the certificate, published on this page and on our COA page rather than packed with orders. View the Melanotan-2 Certificate of Analysis →

How do I buy Melanotan-2?

Add the Melanotan-2 pack size you need to your cart and check out: enter your shipping details, then choose your payment method - cryptocurrency or card - on the next step. The batch Certificate of Analysis (COA) is published on this page, so you can check it before you order.

What payment methods can I use to buy Melanotan-2?

At checkout you can pay by card or crypto. New-U supports BTC and selected stablecoin and network-asset payments across Ethereum, Solana, TRON, Polygon, Base, BNB Chain and Linea. You choose your method after confirming your order.

How fast is shipping, and do you ship worldwide?

Yes - we ship worldwide in discreet, unmarked, tracked packaging; lyophilised peptides are stable through the tracked transit window. Delivery typically takes 6–14 business days, and shipping is free on orders over $350.

Research use only - all claims made on this site are for testing and research use only.

Pharmacokinetics

Half-life ~0.5-1.5 h (IV, rat); functional duration extends 24-36 h subcutaneously Absorption route Subcutaneous; Tmax 0.5-1 h (rodent models) Bioavailability ~4.6% oral (rat, Ugwu et al. 1994); ~100% subcutaneous Metabolism / clearance Proteolytic cleavage; cyclic lactam bridge resists enzymatic degradation vs. linear α-MSH Stability Lyophilised: −20 °C freezer. Reconstituted with bacteriostatic water: refrigerate 1-6 °C, protect from light; stable several weeks. Notes Vss 0.2-0.5 L/kg; CL ~0.3 L/kg/h. Crosses the blood-brain barrier, producing both peripheral and central effects.

Research-Observed Effects

UV-independent cutaneous pigmentation (MC1R-mediated eumelanogenesis)

Reduced caloric intake in rodent obesity models

Increased copulatory behaviour in male rat models (pro-erectile)

Facial flushing and transient sebaceous upregulation

Transient nausea following early doses (tachyphylaxis with repeated dosing)

Darkening of pre-existing nevi and appearance of new pigmented macules

Dosage Ranges (Research Context)

Research-context subcutaneous dosing reported in the literature. Not a clinical recommendation.

Dose Frequency Duration Notes 0.025-0.1 mg Microdose titration (research protocols) 3-7 days Used in some protocols to assess tolerability before escalation 0.25-0.5 mg Once daily (loading) 1-2 weeks Subcutaneous; most commonly reported loading phase in self-experiment literature 0.5-1.0 mg 2-3× per week (maintenance) Ongoing Titrate to desired pigmentation endpoint

Stacking Compatibility

Compatible compounds

PT-141 (bremelanotide): Same melanocortin family; PT-141 is more MC4R-selective for targeted arousal research without layering MC1R pigmentation effects.

GHK-Cu: Complementary dermatological research peptide; copper-binding tripeptide operates via distinct mechanism.

BPC-157: Unrelated cytoprotective mechanism; no known melanocortin interaction in published literature.

Avoid combinations

Other non-selective melanocortin agonists: Additive MC4R activation increases risk of nausea, hypertension, and arousal side effects.

MAOIs / serotonergic agents (research-context): Theoretical interaction through central neuroendocrine pathways; not formally characterised.

Pre-existing atypical melanocytic lesions: MT-2 darkens existing nevi and may obscure dermoscopic surveillance of atypical moles.

Side Effects (Observed in Literature)

Common

Nausea (especially with first 2-3 doses)

Facial flushing / transient vasodilation

Fatigue or drowsiness within 1-2 h of injection

Darkening of existing nevi and freckles

Reduced appetite

Injection-site reactions (erythema, minor swelling)

Rare

Priapism in male subjects

Rhabdomyolysis (isolated case reports with very high doses)

New-onset atypical pigmented macules

Posterior reversible encephalopathy (single case report, 2013)

Dose-dependent

Nausea intensity scales with dose and peak concentration

Pigmentation depth scales with cumulative dose

Central arousal effects scale with single-dose magnitude

Evidence Tier

Overall: Mixed (human + animal)

Mechanism is well-characterised in human and animal receptor pharmacology (Tier 1-2). Pigmentation and arousal endpoints have small human pilot studies (Tier 1). Long-term safety in research subjects is predominantly anecdotal (Tier 3). MT-2 itself is not FDA-approved; bremelanotide (PT-141, closely related) is approved for HSDD.

Tier 1 · Human clinical

Dorr et al. 2004, small human trial of MT-2 + UV combined tanning response (Arch Dermatol).

Wessells et al. 2000, human pilot assessing erectile response to MT-2 (Int J Impot Res).

Bremelanotide (PT-141) Phase III trials, read-across for MC4R pharmacology in humans.

Tier 2 · Animal

Hruby et al. 1995, MT-2 design, rodent receptor binding and in-vivo pigmentation (J Med Chem).

Ugwu et al. 1994, rat pharmacokinetics and oral bioavailability (Pharm Res).

Rodent appetite-suppression and sexual-behaviour models across the 1990s-2000s.

Tier 3 · Anecdotal

Large body of self-experiment reports on pigmentation onset, nausea patterns, and dose titration.

Reports of new atypical nevi and dermatology referrals in unsupervised use.

Certificates, databases & peer-reviewed sources

Last reviewed: 16 June 2026 · New-U Research Compounds

Dorr RT et al. Effects of a superpotent melanotropic peptide in combination with solar UV radiation on tanning of the skin in human volunteers. Arch Dermatol. 2004;140(7):827-35. · 2004 · PMID: 15262696

Hadley ME, Dorr RT. Melanocortin peptide therapeutics: historical milestones, clinical studies and commercialization. Peptides. 2006;27(4):921-30. · 2006 · PMID: 16476498 · DOI: 10.1016/j.peptides.2005.01.029

Hruby VJ et al. Cyclic lactam alpha-melanotropin analogues of Ac-Nle4-cyclo[Asp5,D-Phe7,Lys10] alpha-melanocyte-stimulating hormone (4-10)-NH2 with bulky aromatic amino acids at position 7. J Med Chem. 1995;38(18):3454-61. · 1995 · PMID: 7658432

Ugwu SO et al. Skin pigmentation and pharmacokinetics of melanotan-I in humans. Biopharm Drug Dispos. 1997;18(3):259-69. · 1997 · PMID: 9113347

Wessells H et al. Melanocortin receptor agonists, penile erection, and sexual motivation: human studies with Melanotan II. Int J Impot Res. 2000;12 Suppl 4:S74-9. · 2000 · PMID: 11035391

Habbema L et al. Risks of unregulated use of alpha-melanocyte-stimulating hormone analogues: a review. Int J Dermatol. 2017;56(10):975-80. · 2017 · PMID: 28266027 · DOI: 10.1111/ijd.13585

PubMed: peer-reviewed literature on Melanotan-2

ClinicalTrials.gov: registered studies on Melanotan-2

Melanotan-2: Wikipedia

WebMD: consumer health reference

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Key Characteristics

Cyclic lactam-bridged heptapeptide analog of α-MSH

Non-selective MC1/3/4/5 receptor agonist

Crosses the blood-brain barrier for central effects

D-Phe7 substitution confers protease resistance

Active at sub-milligram subcutaneous doses

Precursor concept that led to FDA-approved PT-141

WADA-prohibited; not approved for human therapeutic use

Research-grade purity: >99% HPLC

Specifications

Molecular Formula C 50 H 69 N 15 O 9 Molecular Weight 1024.18 Da Sequence Ac-Nle-c[Asp-His-D-Phe-Arg-Trp-Lys]-NH 2 Purity >99% (HPLC) Form Lyophilised powder Half-life ~1 h (IV, rodent) Route Subcutaneous injection Solubility Bacteriostatic water or sterile saline Storage Lyophilised: −20 °C freezer. Reconstituted: 1-6 °C, away from light.

About Melanotan-2 (MT-2): Tanning, Libido, Dosage & Melanocortin Research Guide

Melanotan-2 came out of a University of Arizona search for an α-MSH analog that could protect fair-skinned people from UV damage without requiring sun exposure. The cyclic structure and D-Phe substitution solved two problems at once - it made the peptide stable in the bloodstream, and it turned it into a much more potent agonist than the natural hormone it was modelled on.

The non-selective profile is both MT-2's strength and its limitation. It does produce the expected UV-independent pigmentation, but it also activates MC4R in the hypothalamus, which is why research subjects routinely experience nausea, facial flushing, and changes in appetite and libido. That hypothalamic arm of the pharmacology is exactly what inspired the development of the more targeted MC4R agonist bremelanotide (PT-141).

New-U Research Compounds supplies Melanotan-2 as a lyophilised powder at >99% HPLC purity, independently verified by third-party labs. Material is strictly for in-vitro and preclinical research. MT-2 is not approved for human therapeutic use, is a WADA-prohibited substance for competing athletes, and is not a substitute for medically supervised treatment of any condition.

Melanotan II - Wikipedia

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Alpha-Melanocyte-stimulating hormone - Wikipedia

Customer Reviews

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The tracking link made a difference while I waited for Melanotan II. Far better than having no updates.

- George W***** · 26 October 2026

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- Emils P***** · 22 October 2026

Tracking was handy - Melanotan II

The items were packed neatly, not just shoved together. Small detail but I noticed it.

- Ken C***** · 22 October 2026

More in Tanning / Libido

Tanning / Libido Melanotan-1 Afamelanotide (Melanotan I) linear 13-amino-acid α-MSH analogue selectively activates MC1R eumelanogenesis via cAMP-MITF cascade. FDA/EMA-approved for EPP treatment. Melanotan-1 →

Tanning / Libido PT-141 Bremelanotide (PT-141) MC4R agonist studied for hypothalamic sexual arousal pathways via central melanocortin signaling. t½ 2.7 hours SC. Supplied as laboratory research material, not as the approved medicine Vyleesi. PT-141 →

Tanning / Libido Oxytocin Acetate Oxytocin acetate is the acetate salt of the endogenous nonapeptide hormone oxytocin, studied as a reference ligand for the oxytocin receptor in social-cognition, bonding, and smooth-muscle signalling research. Oxytocin Acetate →

Melanotan-2 vs related Tanning / Libido compounds

Compound Price Purity spec COA Melanotan-2 from $81 >99% HPLC 2 published batches Melanotan-1 from $81 >99% HPLC On request PT-141 from $108 >99% HPLC On request

Descriptive catalog comparison for research sourcing decisions - not dosing guidance.

More on Melanotan-2

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Melanotan-2 research guide

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Research this compound

Research guide Melanotan-2 research guide MT-2 binds all four melanocortin receptors, triggering melanin production in the skin and activating appetite and arousal circuits in the… Read the guide →

Analytical evidence View the Melanotan-2 COA Latest published certificate of analysis (purity and identity), dated 2026-04-28. View the COA →

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